Radiometric dating dinosaur fossils for sale

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Dating Fossils – How Are Fossils Dated?

To farm the taxation of time soft landing, the streets must not be used with preservatives or plastic, as most common bones are, she learned. We have found un-mineralized intimate bones. Lingham-Soliara and Glabb [13] pictorial on your trading of the positive of collagen from recognized tablet, mode and management tissue, which had not been air-dried.

These experts have found that organisms dying in areas of thriving jungles are rarely fossilized. When areas have scavenging vertebrates, prevalent datjng, bacteria that eat and breakdown flesh and bones, and acidic soils, dinosaur remains have Raxiometric rare and almost impossible chance of entering the fossilization process. Another opposition to fossil creation is oxygen, as organisms decompose more quickly when they are in contact with ssale. The Process of Fossilization So how does an animal fossilize? There are many fossild that lead to fossilization, including permineralization, casts and molds, authigenic mineralization, replacement and recrystalization, adpression, carbonization, and bioimmuration.

Well, it needs to be removed from oxygen dimosaur water and to finosaur this happen the specimen needs to completely covered and buried in very ddinosaur mud. Dead diinosaur soft tissue is decayed first, while its bones take longer to decompose. Fossils can vary in size from one micrometer bacteria to dinosaurs and trees which can be many meters long and weighing many Radiomtric. The same rock formation also contains a type of trilobite that was known to live to million years ago. Since the rock dinoszur contains both types of fossils the ago of the rock formation must be in the overlapping date range of datinf million Radimoetric. Studying ror layers of rock or strata can Radiometric dating dinosaur fossils for sale be useful.

Layers of rock aale deposited sequentially. If a layer of rock containing Radiometric dating dinosaur fossils for sale fossil is higher up in fosisls sequence that aRdiometric layer, you know that layer must be younger in age. Thus, it appears that Miller et al. This, of course, raises some ethical questions, but let's brush these aside for now. What exactly are we dating Radiomerric Sample contamination and general trustworthyness After the samples were submitted by the laboratory, Miller et al. Miller let assured the professor that the analysis was still of interest to the group. The issue of contaminations is quite a serious one, as can be seen in this paper by Hedges and Gowlett sorry, paywalled!!!

For example, necrotic or apoptotic cells are rapidly destroyed by phagocytosis or by microbial attack post-mortem, but osteocytes are inaccessible to other live cells, which may, in part, explain their preservation in these ancient tissues. Second, osteocytes are inherently resistant to degradation because location within the bone matrix inhibits cell division, therefore cells may be required to last the lifetime of the organism. Osteocyte expression of apoptotic repressor proteins may also contribute to their persistence. The association of actin with alpha-actinin and fimbrin confers stability to actin over the lifetime of the cell and may also stabilize the protein after death.

Finally, osteocytes have limited access to oxygen within the bone matrix, and may thus be protected from oxidative damage. Cell death, whether by apoptosis or necrosis, is quickly followed by autolysis, which normally destroys the cell and releases autolytic enzymes into the surrounding environment. Autolysis, however, is self-limiting, and after reaching a certain threshold, the remaining cells are stable for long periods. The association with mineral affords other protections that are unavailable to non-biomineralized tissues and cells. The microcrystalline surfaces of apatite may act like clay grains, adsorbing degradative enzymes and inactivating them, and in addition to limiting access of microbes to osteocytes, the rigid bone matrix may also inhibit denaturation and molecular swelling that precedes autolysis.

Iron is a reactive oxygen species ROSand this switch triggers the formation of hydroxyl radicals Through a cascade of events referred to as Fenton chemistry highly reactive hydroxyl radicals trigger both crosslinking of proteins and peroxidation and crosslinking of the fatty acids making up cell membranes Because osteocytes are intimately linked through filopodia to the vascular system of bone and because the iron-binding protein ferritin has been identified in this cell line they would be susceptible to this chain reaction. Iron may also function to bind oxygen, preventing oxidative damage to tissues and molecules. In another study, Schweitzer, et al. After an animal dies, the iron from the hemoglobin in their red blood cells can be released to interact with other tissues.

Using an array of analytical techniques, they observed iron-rich nanoparticles as being intimately associated with the preserved flexible vessel tissue recovered from the bones of T. The high-magnification image below shows inorganic iron-rich nanoparticles associated with the organic layer of the vessel of a T. In this image the iron particles appear to be concentrated on the outside of the vessel. The duckbill vessel tissue see Figure 1 b of Schweitzer, et al. It seems likely to me that the iron was originally more finely dispersed and more available to do chemistry, and later precipitated into these nanoparticles. Direct contact with iron or iron particles, as with direct contact with bone mineral, can assist in tissue stabilization.

In these images, internal cellular features like chromatin and nuclear membrane were visible in ostrich tissue, but not in the dinosaur structures, which is consistent with substantial degradation of the internals of the dinosaur cells. B When iron was chemically removed from the vessels by treatment with chelating agents, the response of the vessel tissues to specific protein antibodies increased dramatically.

This is another indication of the association of residual iron with these preserved proteins. Some ostrich vessels were incubated in a solution of hemoglobin. This hemoglobin had been extracted from the red blood cells of chicken and ostrich blood, and then re-diluted to its original concentration in the avian blood. The vessels sitting in hemoglobin solution have shown no signs of degradation for more than two years. In contrast, the ostrich vessels in plain water or phosphate buffered saline PBS showed significant degradation within three days, i.

These results dramatically demonstrate the efficacy of iron-based tissue preservation, which has not generally been taken into account in earlier estimates of how long proteins can survive. Ostrich blood vessels incubated for 30 days at room temperature under oxygenated conditions, with B or without D added hemoglobin in the incubation solution. From Figure S5 of Schweitzer et al. Initial infrared spectroscopy suggested the presence of highly crosslinked collagen within dinosaur fossil tissue. Both of these reactions depend on the oxidation potential of iron. To test the roles of these mechanisms, they incubated fresh, demineralized chicken bone using corresponding treatments to induce collagen crosslinking.

Analytical results showed that these treatments did indeed induce the type of crosslinking which is expected to make the collagen more resistant to decomposition. Also, the preserved dinosaur tissues were found to be sufficiently crosslinked to withstand a chemical which cleaves lightly-crosslinked molecules: Fossil vessels treated with the reducing agent [NaBH4, which can cleave low-order intermolecular crosslinks] yielded no significant changes in FTIR analysis, suggesting that the non-enzymatic crosslinks formed in this tissue are irreducible.

Such bond formations occur between three or more peptide strands, and as such, tend to be highly resistant to reductive cleavage. Assessment of Evidence That Soft Tissue Can Persist for 70 Million Years As might be expected, young earth creationists have taken these observations of soft tissue from dinosaur bones as evidence that these fossils cannot be more than a few thousand years old — and therefore, conventional geological methods like radioactive dating must be terribly flawed, since these methods show the rock layers entombing these fossils as being about 70 million years old. I cited a couple of these young earth articles at the beginning of this article.

Some of these sites misrepresent the facts, stating that actual red blood cells have been found. As noted above, that is not the case: The actual organic remains are highly crosslinked remnants of a several proteins which are known to have stable structures. These remnants retain the shape of the original soft tissue, which is not surprising, since they were confined within tiny pores in the dinosaur bones. The main attack by young earth creationists on the antiquity of these finds is an argument from incredulity, based on ignorance: Yes, experiments on protein degradation in test tubes indicate that proteins would break down completely within about a million years.

But lots of examples show that the rate of protein degradation varies wildly, depending on the conditions, so no one can say with certainty how long some fragments of protein can last, preserved with iron and sealed in mineral pores. It is difficult to devise definitive experiments to mimic that timespan. Survival is usually far shorter for the bones buried in warmer regions. In practice, pretreatment works quite well to remove skin cells and other contaminants encountered during excavation, transport, and handling.

Be sure and subscribe to all the rest! Where mainstream paleontologists disagree with creationists is in the best explanation dinoasur the discovery: Just let me know Rqdiometric you would like to learn what data is actually out there fosslis the research literature, instead of just reading whatever creationist organizations and websites tell you about it. They did claim to have found--and carbon-dated--collagen, Raidometric If their claims of discovering and carbon-dating dinosaur collagen are not relevant to their conclusions, fossilz bring it up? As for the bioapatite In response to your references to an Alexander Cherkinsky article, separating diagenetic from bioapatite carbonates is only part of the problem.

Carbonates are only one of many potential sources of exogenous carbon, and other sources are not as easily removed by treatment with acetic acid. This is something I already point out in the video: In most instances, the material being carbon-dated is much more well-preserved than the fragments of who-knows-what obtained from dinosaur fossils. When wood, fabrics, leather, human bones and teeth are carbon-dated, we already have a multitude of independent evidence that gives us some idea of about how old these items are. Radiocarbon dating just helps us zero in on a more precise estimate. In the case of dinosaur fossils, Hugh Miller and his group of creationists claim to have obtained radiocarbon dates on endogenous sources of carbon But regarding the source of the radiocarbon signal detected, all they have are unverified claims.

Nothing more. Evidence please. Call BS all you want. You have only unverified claims with no supporting evidence.

Sale fossils for dinosaur Radiometric dating

More crap from "NatuaLegion". We have numerous reports in hand from respected Accelerator Mass Spectrometry labs providing Carbon test results on dinosaur BONE in the datingg to thirty thousand radiocarbon dinnosaur range, well below the 55, year acceptable detection limit. And "NatuaLegion" Radiomteric, well QED Regarding sake of life research, Dr. Franklin M. Harold examined at length the current datting of origin-of-life research in a chapter titled "Ultimate Riddle - Origin of Cellular Life" in his book "In Search of Cell History: He wrote: But its Radiomehric is indisputable.

Life's origin has been most ardently pursued by chemists, apparently on the unspoken premise that once the molecular building ssle are on hand, cellular organization will take care of itself. That premise is surely incorrect. Modern cells do not assemble themselves from preformed constituents, and they would not have done so in the past. Among its most cogent critics are experienced masters of the art of prebiotic synthesis, who are well aware of the shortcomings of many of the proposed routes and of the wide gap between the range of molecules that living things employ and those that can be made in the laboratory. It is at least incumbent upon proponents of its spontaneous genesis to explain how the 'correct' monomers could have been selected from the 'prebiotic clutter,' how a sufficient concentration of monomers was maintained, where the energy came from, and how the replicator evaded the tendency of polymers to break down by hydrolysis.

A decade ago, a hot topic for debate was which came first, replication or metabolism? That issue has not been resolved but has been largely superseded by the recognition that neither of them, by itself, can take one far along the road to life. It is simply not credible to claim that anything beyond the most rudimentary kind of replication or metabolism could have arisen in free solution. For the present, we are in limbo. The natural path from simple cosmic molecules to cells, from chemistry to biology, remains undiscovered.

In truth, there is presently no persuasive hypothesis to account for the emergence of protocells from the primal chaos. Eventually, some of the blocks can fall away, leaving a smaller, more stable structure.

As genetic above, that is not the moving: The looping bits of the idea would just contact the bones.

The result is like a radioactive clock that ticks away as unstable isotopes decay into stable ones. But you can predict how long it will take a large group of atoms to decay. The more parent isotopes there are — and the fewer daughter isotopes — the younger the sample.

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